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In order to accomplish this, we utilize a mouse model with the chronic autoimmune inflammatory disorder, systemic lupus erythematosus (SLE). SLE is an ideal experiment model to examine the link between inflammation, the kidneys, and hypertension because its origins are rooted in immune system dysfunction and patients with SLE hve prevalent hypertension and renal disease.
Some of the ongoing studies in the laboratory are focused on renal hemodynamic changes during SLE, the role of specific inflammatory cytokines in SLE hypertension, neural-immune interactions that promote hypertension and renal disease, and the regulation of blood pressure and renal injury by sex steroids during SLE.
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